BioMed Research International
Volume 2020 (2020), Article ID 9175369, 2 pages
https://doi.org/10.1155/2020/9175369

Corrigendum

Corrigendum to “Peripheral Blood Mononuclear Cells as a Laboratory to Study Dementia in the Elderly”

Beatrice Arosio,1,2 Claudio D'Addario,3,4 Cristina Gussago,² Martina Casati,¹ Enzo Tedone,² Evelyn Ferri,² Paola Nicolini,¹ Paolo D. Rossi,¹ Mauro Maccarrone,5,6 and Daniela Mari1,2

¹Geriatric Unit, Fondazione Ca’ Granda, IRCCS Ospedale Maggiore Policlinico, Via Pace 9, 20122 Milan, Italy
²Geriatric Unit, Department of Medical Sciences and Community Health, University of Milan, Via Pace 9, 20122 Milan, Italy
³Faculty of Bioscience and Technology for Food, Agriculture and Environment University of Teramo, Piazza Aldo Moro 45, 64100 Teramo, Italy
⁴Department of Clinical Neuroscience, Karolinska Institutet CMM L8:01, 17176 Stockholm, Sweden
⁵European Center for Brain Research, Santa Lucia Foundation, IRCCS, Via del Fosso di Fiorano 64, 00143 Rome, Italy
⁶Center of Integrated Research, Campus Bio-Medico University of Rome, Via Alvaro del Portillo 21, 00128 Rome, Italy

Correspondence should be addressed to Beatrice Arosio; beatrice.arosio@unimi.it

Received 21 July 2020; Accepted 18 August 2020; Published 11 September 2020

Copyright © 2020 Beatrice Arosio et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

The article titled “Peripheral Blood Mononuclear Cells as a Laboratory to Study Dementia in the Elderly” [1] was found to contain a substantial amount of material from previously published articles including the following sources:

(i)Manraj S. Bhamra and Nicholas J. Ashton, “Finding a pathological diagnosis for Alzheimer’s disease: Are inflammatory molecules the answer?: Proteomics and 2DE”, Electrophoresis, 2012. 10.1002/elps.201200161. [2] (not cited)(ii)Kelly M. Bakulski, Laura S. Rozek, Dana C. Dolinoy, Henry L. Paulson and Howard Hu, “Alzheimer’s Disease and Environmental Exposure to Lead: The Epidemiologic Evidence and Potential Role of Epigenetics”, Current Alzheimer Research (2012) 9: 563. 10.2174/156720512800617991. [3] (cited as reference [75])(iii)Cristina Gussago, “Il Recettore Adenosinico A2a Come Possibile Biomarcatore Nella Diagnosi Differenziale Delle Demenze Nell’anziano”, Università Degli Studi Di Milano. 10.13130%2Fgussago-cristina_phd2014-03-10. [4] (not cited)(iv)B. Arosio, A. Bulbarelli, S. Bastias Candia, E. Lonati, L. Mastronardi, P. Romualdi, S. Candeletti, C. Gussago, D. Galimberti, E. Scarpini, B. Dell Osso, C. Altamura, M. Maccarrone, L. Bergamaschini, C. D Addario, D. Mari. “Pin1 Contribution to Alzheimer s Disease: Transcriptional and Epigenetic Mechanisms in Patients with Late-Onset Alzheimer s Disease”, Neurodegenerative Diseases, 2012.10.1159/000333799. [5] (cited as reference [66])(v)K. Ando, P. Dourlen, A. V. Sambo et al., “Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker,” Neurobiology of Aging, vol. 34, no. 3, pp. 757–769, 2013. 10.1016/j.neurobiolaging.2012.08.004. [6] (cited as reference [69])

The authors apologise in particular for not acknowledging the work of Bhamra and Ashton (2012).

References

B. Arosio, C. D'Addario, C. Gussago et al., “Peripheral blood mononuclear cells as a laboratory to study dementia in the elderly,” BioMed Research International, vol. 2014, Article ID 169203, 14 pages, 2014.

M. S. Bhamra and N. J. Ashton, “Finding a pathological diagnosis for Alzheimer's disease: are inflammatory molecules the answer?” Electrophoresis, vol. 33, no. 24, pp. 3598–3607, 2012.

K. M. Bakulski, L. S. Rozek, D. C. Dolinoy, H. L. Paulson, and H. Hu, “Alzheimer’s disease and environmental exposure to lead: the epidemiologic evidence and potential role of epigenetics,” Current Alzheimer Research, vol. 9, no. 5, pp. 563–573, 2012.

C. Gussago, Il Recettore Adenosinico A2a Come Possibile Biomarcatore Nella Diagnosi Differenziale Delle Demenze Nell’anziano, UniversitàDegliStudi Di Milano, 2014.

B. Arosio, A. Bulbarelli, S. Bastias Candia et al., “Pin1 contribution to Alzheimer’s disease: transcriptional and epigenetic mechanisms in patients with late-onset Alzheimers disease,” Neurodegenerative Diseases, vol. 10, no. 1-4, pp. 207–211, 2012.

K. Ando, P. Dourlen, A.-V. Sambo et al., “Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker,” Neurobiology of Aging, vol. 34, no. 3, pp. 757–769, 2013.